ESIS

Energy signaling in the stress response

 Coordinatore FUNDACAO CALOUSTE GULBENKIAN 

 Organization address address: AVENIDA DE BERNA 45A
city: LISBOA
postcode: 1000

contact info
Titolo: Prof.
Nome: Antonio
Cognome: Coutinho
Email: send email
Telefono: -214407550
Fax: -214410501

 Nazionalità Coordinatore Portugal [PT]
 Totale costo 75˙000 €
 EC contributo 75˙000 €
 Programma FP7-PEOPLE
Specific programme "People" implementing the Seventh Framework Programme of the European Community for research, technological development and demonstration activities (2007 to 2013)
 Code Call FP7-PEOPLE-IRG-2008
 Funding Scheme MC-IRG
 Anno di inizio 2008
 Periodo (anno-mese-giorno) 2008-12-01   -   2011-11-30

 Partecipanti

# participant  country  role  EC contrib. [€] 
1    FUNDACAO CALOUSTE GULBENKIAN

 Organization address address: AVENIDA DE BERNA 45A
city: LISBOA
postcode: 1000

contact info
Titolo: Prof.
Nome: Antonio
Cognome: Coutinho
Email: send email
Telefono: -214407550
Fax: -214410501

PT (LISBOA) coordinator 0.00

Mappa


 Word cloud

Esplora la "nuvola delle parole (Word Cloud) per avere un'idea di massima del progetto.

responses    stress    snf    energy    protein    mode    snrk    kinase    signaling    pathway    mechanisms    despite    plants    cell   

 Obiettivo del progetto (Objective)

'Plants are constantly confronted by multiple types of nutritional, abiotic and biotic stress. Often associated with stress is a reduction in photosynthesis and/or respiration, which in turn results in energy deprivation and ultimately in growth arrest. Despite their distinct origin and mode of perception, different stresses trigger similar downstream responses that include largely overlapping patterns of gene expression. Our work has shown that this general stress transcriptome is partly regulated by the evolutionarily conserved energy sensor protein kinases, SNF1 (sucrose non-fermenting1) in yeast, AMPK (AMP-activated protein kinase) in mammals and SnRK1 (Snf1-related kinase1) in plants. Upon sensing the energy deficit associated with stress, SnRK1 triggers extensive transcriptional changes that contribute to restoring metabolic and energy homeostasis, promoting cell survival and allowing the elaboration of longer-term responses for adaptation, growth and development. Despite the importance of the uncovered energy signaling pathway, virtually nothing is known regarding its mode of operation. Using a combination of cell-based assays, functional genomics, bioinformatics, mutant screens and genetics, this proposal seeks to gain insight on the regulatory mechanisms that govern SnRK1 action and to further dissect this signaling pathway through the identification of novel components. Elucidation of these mechanisms will contribute to understanding how stress resistance is established and how plant growth and development are finely orchestrated by the environment.'

Altri progetti dello stesso programma (FP7-PEOPLE)

COGEPRBN (2008)

Molecular characterization of the PrBn locus and some other QTLs controlling homeologous recombination in Brassica napus

Read More  

COARSE ANALYSIS (0)

Analytic problems in Coarse Geometry and Geometric Group Theory

Read More  

DICE (2011)

Direct Imaging of Extrasolar Planets from LBT and VLT to E-ELT

Read More