FUNGI-PATHNCODE

The genomic basis of emerging fungal pathogenicity

 Coordinatore FUNDACIO CENTRE DE REGULACIO GENOMICA 

 Organization address address: CARRER DOCTOR AIGUADER 88
city: BARCELONA
postcode: 8003

contact info
Titolo: Mr.
Nome: Conangla
Cognome: Joan
Email: send email
Telefono: 34933160234
Fax: 34933969983

 Nazionalità Coordinatore Spain [ES]
 Totale costo 230˙480 €
 EC contributo 230˙480 €
 Programma FP7-PEOPLE
Specific programme "People" implementing the Seventh Framework Programme of the European Community for research, technological development and demonstration activities (2007 to 2013)
 Code Call FP7-PEOPLE-2010-IEF
 Funding Scheme MC-IEF
 Anno di inizio 2012
 Periodo (anno-mese-giorno) 2012-01-01   -   2013-12-31

 Partecipanti

# participant  country  role  EC contrib. [€] 
1    FUNDACIO CENTRE DE REGULACIO GENOMICA

 Organization address address: CARRER DOCTOR AIGUADER 88
city: BARCELONA
postcode: 8003

contact info
Titolo: Mr.
Nome: Conangla
Cognome: Joan
Email: send email
Telefono: 34933160234
Fax: 34933969983

ES (BARCELONA) coordinator 230˙480.00

Mappa


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species    fungal    genes    selective    pathogenic    regulatory    human    pathogens    genomic    noncoding   

 Obiettivo del progetto (Objective)

'In the study of human pathogens most of the attention has been given to viral and bacterial species, and fungi have been largely neglected. To date there is no broad, large-scale census of the genomic elements shared by fungal pathogenic species. Also, no large-scale scan for selective pressure variability has been undertaken, even though all the elements are in place to do so. There has neither been a multi-species search for conserved noncoding elements that may be key to explaining the regulation of crucially important functional elements and networks. The overall goal of this study is to gain insight on the evolution of the genomic elements involved in fungal human pathogenicity through the analysis of the selective constraints acting on coding and noncoding sequences. For this purpose we will combine genome sequencing, transcriptomics (RNA-seq), comparative genomics and evolutionary analyses. This project will throw light on fundamental questions about the key genes, regulatory elements and phenotypic traits that are involved in infection or how emergent, opportunistic pathogens originate relatively rapidly from non-pathogenic ancestors. Finally, the list of identified relevant genes and regulatory elements can provide much needed targets for developing pharmaceutical and therapeutic strategies.'

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